Experimental allergic encephalomyelitis (EAE) is considered as a primary demyelinated disease,characterized by wide demyelination and relatively reserved axons for long time. Not until the end of last century is it proved that there are axon injures in the EAE focus. And people do extensive research to explore measures to help myeline and axons regenerate,but make little progress. At present, there is no effective therapy.
Generally, the therapy of EAE is as the following:(1) to activate the intri...
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