Background and Objective:
Congenital heart disease, especially cyanotic cardiac defects which result in chronic hypoxia in the neonate until operative correction is achieved, remains a major cause of death in infancy. The effects of chronic hypoxia on neonatal myocardial metabolic adaptation have not been well defined. In the heart, AMP-activated protein kinase (AMPK) orchestrates the regulation of energy generating and consuming pathways by regulating many metabolic pathways, and it’s the key...
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