【摘要】 目的 :探讨门脉高压性胃病的发病机制。方法 :用血气分析法观测 43例门脉高压症患者股动脉血、胃网膜右动、静脉胃壁支血气变化、胃网膜右静脉压力。结果 :门脉高压症患者的股动脉血PO2 低于对照组 ;门脉高压症患者胃网膜右动、静脉胃壁支血氧分压差低于对照组 ,胃网膜右静脉压力高于对照组 ,胃壁支静脉血pH和HCO-3 低于对照组和同组肘静脉血。并有门脉高压性胃病者这些变化更为显著。结论 :门脉系压力增高、胃粘膜下动静脉短路开放、胃粘膜缺血缺氧和胃壁局部中毒 ,是门脉高压性胃病的发病机制之一。更多还原

【Abstract】 Objective: To study the mechanisms of portal hypertensive gastropathy.Methods: With blood gas analysis to determine the blood gas changes of right gastroepiploic arteriovenous parietal branches and the pressure on right gastroepiploic vein in 43 cases of portal hypertension.Results: ① PO 2 of femoral artery in those with portal hypertension was lower than in control group.② PO 2 of right gastroepiploic arteriovenous parietal branches in those with portal hypertension was lower than in control group.Pressure on right gastroepiploic venous branch was higher than that in the control group.PH and HCO - 3 of parietal venous branch were lower than in control group and cubital vein in the same group.The changes above were more obvious in the patients complicated with portal hypertensive gastropathy.Conclusion: The opening of gastric submucosal arteriovenous shunt by increased portal pressure causing gastric mucosa ischemia or hypoxia and local gastroparietal acidosis is one of the mechanisms of portal hypertensive gastropathy.更多还原