【摘要】 目的与血管造影对照探讨急性脑梗死磁共振液体衰减反转恢复序列(fluid-attenuated inversion recovery,FLAIR)大脑中动脉高信号血管征(hyperintense vessel sign,HVS)的形成机制。方法回顾性分析15例急性大脑中动脉闭塞6 h内患者的磁共振FLAIR序列及血管造影图像,并以血管造影分析其血流动力学特征。结果 15例急性大脑中动脉闭塞患者HVS出现率为100%,颈内动脉造影及微导管局部造影显示M1段充盈缺损,M1段以远侧支逆流显影、造影剂滞留。结论急性脑梗死HVS形成的机制可能为近端血管内血栓形成,远端血流缓慢的侧枝循环。更多还原

【Abstract】 Objective To discuss the HVS formation mechanism based on magnetic resonance FLAIR sequence and angiography for acute cerebral infraction patients. Methods Within 6 h,15 cases of acute middle cerebral artery occlusion received magnetic resonance FLAIR sequence and angiography images. The hemodynamic characteristics was analyzed based on angiography. Results For 15 cases of acute middle cerebral artery occlusion,the incidence of HVS was 100%. According to the internal carotid artery angiography and micro catheter local angiography,the filling defect was presented at M1 segment. The collateral M1 segment had the countercurrent imaging and retention of contrast medium. Conclusion For acute cerebral infraction patients,the formation mechanism of HVS may be attributed to the proximal intravascular thrombosisand slow collateral circulation of distal blood flow.更多还原