节点文献
静脉麻醉药与脑保护受体机制
Intravenous anesthetics and the receptor mechanism of neuroprotection
【摘要】 兴奋毒性可能是造成神经元死亡的“最后公路”。已知静脉麻醉药 ,如非竞争性NMDA受体拮抗剂 (氯胺酮 )和GABA受体模拟剂 (丙泊酚 )有神经保护作用 ,该文就它们的神经保护作用的受体机制研究加以综述
【Abstract】 The excitotoxicity may be the last public way by which neurons were led to death. It has been shown that intravenous anesthetics, such as non-competitive NMDA antagonists (ketamine) or GABAmimetics (propofol) have neuroprotection. The receptor mechanism of neuroprotection by intravenous anesthesia was reviewed in this essay.
【关键词】 兴奋性氨基酸;
NMDA;
脑保护;
静脉麻醉药;
【Key words】 excitatory amino acid; NMDA; brain protection; intravenous anesthetics;
【Key words】 excitatory amino acid; NMDA; brain protection; intravenous anesthetics;
- 【文献出处】 上海第二医科大学学报 ,Acta Universitatis Medicinalis Secondae Shanghai , 编辑部邮箱 ,2004年11期
- 【分类号】R614.24
- 【被引频次】3
- 【下载频次】246