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心肌缺血致室性心动过速的发生机制的探讨

AN EXPLORATION ON MACHANISM OF VENTRICULAR TACHYCARDIA DUE TO MYOCARDIAL ISCHEMIA

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【作者】 姜晓霞张哲孙静于晔赵丽

【Author】 JANG Xiaoxia , HANG Zhe , SUN Jing , et al Department of Function, First Affiliated Hospital of Medical College of Shihezi University, Shihezi, 832008

【机构】 新疆石河子大学医学院一附院功能科新疆石河子大学医学院一附院功能科

【摘要】 目的:探讨冠心病患者心肌缺血致室性心动过速的发生机制。方法:对冠心病患者心肌缺血(MI)时伴室性心动过速的18例病人(Ⅰ组)与无室速的37例病人(Ⅱ组)进行24h动态心电图监测。结果:Ⅰ与Ⅱ组间每天MI总阵次和总时间有非常显著性差异(P<0.01),Ⅰ组全程持续性MI高于Ⅱ组(P<0.05)。Ⅰ组中伴室速发作的MI持续时间,最大ST压低幅度高于无室速的MI(P<0.01),Ⅰ组3级以上室早总发生率明显高于Ⅱ组(P<0.01)。结论:冠心病患者MI是导致室速的主要病理生理基础,积极治疗MI是预防室速发生的主要措施。

【Abstract】 Objective: To explore the mechanism of ventricular tachycardia due to myocardial ischemia(MI) on patients with coronary heart disease. Methods: 24 hours dynamic electrocardiogram were monitored in 18 patients with coronary heart disease who have the symptom of MI with ventricular tachycardia (G Ⅰ ) and 37 patients with coronary heatr disease without ventricular tachycardia (G Ⅱ ) . Results: There is a marked difference between GⅠ and GⅡ in the frequency and duration of MI ( P < 0.01 ) . The whole course of MI of G Ⅰ is longer than that of GⅡ ( P < 0.05) . In G Ⅰ , MI for the patients wiht coronary heart disease and ventricular tachycardia lasting time and the lowering range of ST is higher than M Ⅰ OF GⅡ . The rate of occurrence of ventricular premature beat of G Ⅰ is higher than that of GⅡ ( P < 0.01) . Conclusion: MI for patients with coronary heart disease is the mam pathological and physiological cause of ventricular tachycardia, so treatment for MI can decrease the occuring rate of ventricular tachycardia.

  • 【分类号】R541.71
  • 【被引频次】13
  • 【下载频次】98
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