【作者】
Katsuya Tanaka；
Lynda M.Ludwig B.S；
Judy R.Kersten；
Paul S.Pagel；
David C.Warltier；
李军；
桂波；
【机构】
Medical College of Wisonsin,Medical College of Wisconsin；
the Clement J. Zablocki Veterans Affairs Medical Center,Milwaukee,Wisconsin.,Medical College of Wisconsin；
the Clement J.Zablocki Veterans Affairs Medical Center,Milwaukee,Wisconsin；
and Department of Biomedical Engineering,Marquette University,Milwallkee,Wisconsin,徐州医学院江苏省麻醉学重点实验室,徐州医学院江苏省麻醉学重点实验室 M.D Research Fellow,Graduate Student,M.D Professor of Anesthesiology and Pharmacology ang Toxicology,M.D.Ph.D Professor of Anesthesiology and Biomedical Engineering and Director of Cardiac Anesthesia,M.D. Ph.D Professor of Anesthesiology,Biomedical Engineering,Pharmacology and Toxicology,and Medicine (Division of Cardiovascular Diseases) and Senior Vice Chairman of Anesthesiology；
【摘要】 <正> 日渐增多的证据表明,吸入麻醉药可保护心肌免受可复性或不可复性缺血性损伤。探讨吸入麻醉药介导抗缺血性效应的机制近来备受关注,由于吸入麻醉药可明显影响心血管功能,如降低动脉及冠脉灌注压、剂量依赖性抑制心肌收缩、舒张冠脉、影响电生理、不同程度调节自主神经系统活性等,使得机制的探讨较为困难。因此心肌氧供需关系的适度更多还原
