【摘要】 目的　探讨正常人和感音性聋患者声反射敏化作用特点及其作用机制。方法　选用 30名 ( 6 0耳 )正常人和 15耳早期梅尼埃病患者、34耳老年性聋、2 7耳突发性聋患者作为测试对象 ,以单耳双声高频敏化低频方法测试声反射敏化反应。结果　正常人 6 0耳均能引出声反射敏化作用 ,单耳双声敏化法敏化值为 11.4 8± 7.6 9dB ,双耳双声敏化值为 6 .2 7± 6 .32dB ;15耳早期梅尼埃病敏化值为 6 .5 3± 5 .77dB ;34耳老年性聋敏化值为 5 .35± 5 .77dB ;2 7耳突发性聋敏化值为 5 .6 3± 5 .0 8dB。病变组与正常听力组间比较P <0 .0 1,病变组间比较P >0 .0 5。结论　正常人均可引出声反射敏化作用 ,感音性聋敏化值较正常人为小 ,敏化作用机制和耳蜗功能损害相关更多还原

【Abstract】 Objective To explore the characteristics of high-frequency sensitization of the acoustic reflex in sensorineural hearing loss.Methods 30 normal-hearing subjects and 76 sensorineural hearing-impaired individuals were included,both the facilitating and the reflex-eliciting signals were employed in this study.Results Both acoustic reflex and sensitization could be elicited in 30 normal-hearing subjects.The value of sensitization obtained in the present study was 11.48±7.69 dB at monotic mode and 6.27±6.32 dB at dichotic mode in normal-hearing subjects, 6.35±5.77 dB in 15 Menieres disease patients, 5.35±5.77 dB in 34 presbycusis, 5.63±5.08 dB in 27 sudden deafness, respectively. There was significant difference between the normal and sensorineural hearing loss groups ( P <0.01), whereas the differences among the sensorineural hearing-impaired groups were not statistically significant( P >0.05).Conclusion Threshold difference in sensorineural hearing loss was lower than that in normal-hearing subjects, the mechanism was related to cochlear lesion.更多还原