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低血钾软病的肾小管功能观察及失钾机理的探讨
Observations of the Function of Renal Tube and theDiscussion of the Mechanism of PotassiumLoss in Hypokalemic Flabbiness Disease
【摘要】 <正> 近10多年来,我国部分棉产区发现一种地方性、流行性低血钾麻痹症。我们曾提出本病有类似肾小管酸中毒的改变,发病可能与肾小管受损有关,并称其为低血钾软病(以下简称软病)。为进一步探讨失钾机理,1980年对60例软病患者作了肾小管功能等检查,并以同期26例低血钾周期性麻痹(以下简称周麻)作对照。
【Abstract】 We have performed a series of examinations of renal tubular function in 60 patients with "hypokalemic flabbiness disease" (HFD) and a control group of 26 cases with periodic paralysis. The results in HFD group were all abnormal, but in the latter were all normal. In most cases of HFD. the results of ammonium chloride loading test were abnormal. The CO2 combining power of 23 patients with HFD at paralytic period was determined, 9 cases were below 41 vol%. Urine potassium of 27 patients with HFD was determined and the results of 23 were compatible with renal potassium loss. We believe that HFD may be caused by certain substance which is harmful to the renal tube and thus induces renal tubular acidosis and renal potassium loss.
- 【文献出处】 武汉医学院学报 , 编辑部邮箱 ,1982年02期
- 【被引频次】3
- 【下载频次】16